Gastritis is the inflammation of the stomach(gastric) mucosa. The inflammation may be localized or scattered throughout the stomach lining.

Classification of Gastritis

Acute gastritis. It is mostly transient and self-limiting in nature; lasting for several hours to a few days and its symptoms appear rapidly. It may be referred to as erosive or non-erosive depending on the effects of gastric mucosa.

Chronic gastritis. It normally results from recurring episodes of acute gastritis and is usually classified as type A Type B or atrophic

Causes of Gastritis

Pathophysiology

The stomach wall is usually protected from corrosion by hydrochloric acid and pepsin by a mucosal barrier. This barrier is produced by prostaglandins.

An impaired mucosal barrier allows the corrosive hydrochloric acid, pepsin and other irritating agents to come into contact with the gastric mucosa resulting inflammation.

This causes the gastric mucosa to become edematous and hyperemic, congested with fluid and blood and to undergo superficial erosion. The resulting injury is exacerbated by the release of histamine and stimulations of the vagus nerve.

Superficial ulceration may occur as a result of erosive diseases and may lead to hemorrhage.

In chronic gastritis, persistent or repeated acute gastritis leads to chronic inflammatory changes and eventual atrophy of the gastric tissue. The progressive gastric atrophy from chronic mucosal injury decreases the amount of or destroys the parietal cells hence the source of intrinsic factor is lost. Intrinsic factor is critical for the absorption of vitamin B12. When Vitamin 12 is depleted pernicious anemia results.

Clinical Manifestations

In acute gastritis, the patient will manifest the following;

In chronic gastritis, the patient will manifest the following;

Assessment and Diagnostic Findings

Medical Management

Nursing Management

Assessment

Interventions

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